Pathophysiology of Pharyngitis
Transmission is by droplet. Germs infiltrate the epithelial layer, then the epithelium is eroded, then the superficial lymphoid tissue inflammatory reaction occurs with the damming of polymorphonuclear leukocyte infiltration. In the early stages there is hyperemia and edema and increased secretions. Serous exudate at first, but tends to become thickened and dry and can be attached to the pharyngeal wall. By hyperemia, blood vessel walls of the pharynx to the width. Blockage forms a yellow, white, or gray present in follicles or lymphoid tissue. It appears that the lymphoid follicles and blotches on the posterior pharyngeal wall or located more laterally to become inflamed and swollen causing inflammation of the throat or pharyngitis.
